The peak DMAA concentration for all subjects was observed within 3–5 hours following ingestion and was very similar across subjects, with a mean of ~70 ng∙mL-1. Heart rate, blood pressure, and body temperature were largely unaffected by DMAA treatment.
But here’s what I find interesting:
These findings indicate a consistent pattern of increase across subjects with regards to peak DMAA concentration, with peak values approximately 15–30 times lower than those reported in case studies linking DMAA intake with adverse events.
So… what were those “adverse events” all about? Random gene mutation where you can’t handle DMAA… or maybe… they weren’t following the label? Hmmmm…